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P values based on two-tailed, matched-pair t-tests were calculated for the comparisons between the 6-month and month values in group 1 and group 2.

In addition, for each response variable the 6-month value was subtracted from the month value to represent the change in each subject.

P values based on two-tailed, unequal-variance, independent-sample t-tests were then calculated for the comparison of the changes in response variables between groups 1 and 2.

All the men remained healthy, and none had any changes in the results of differential blood count, urinalysis, blood-chemistry profile, chest radiography, electrocardiography, or echocardiography during the month protocol.

Eight of the 12 men in group 1 required no adjustment in their initial dose of growth hormone. Two required an upward adjustment of 25 percent, and two required a downward adjustment of 25 percent.

The mean plasma IGF-I concentration in group 2 remained in the range of to U per liter throughout the base-line and treatment periods. There was no significant change in weight in either group.

In group 1, several response variables had changed significantly after 12 months. Lean body mass and the average density of the lumbar vertebrae increased by 8.

The sum of skin thicknesses at four sites increased 7. The small average change in lumbar vertebral bone density only 0.

The bone density of the radius and proximal femur and the ratio of the height of the alveolar ridge to total mandibular height did not change significantly.

In group 2 none of these variables changed significantly. The 21 men studied were representative of the approximately one third of all men 60 to 80 years old who have plasma IGF-I concentrations of less than U per liter as compared with a range of to U per liter in healthy men 20 to 40 years old.

Furthermore, our entry criteria focused the study on an overtly healthy subgroup of older men. In the absence of obesity, 4 below-normal weight, 20 or liver disease, 21 a plasma IGF-I concentration of less than U per liter in older men generally signifies that they secrete very little growth hormone.

We did not do this, but Ho et al. Most of the IGF-I plasma is bound to these proteins, but their concentrations vary little in healthy people who eat a normal diet.

In the 12 men in group 1, initially low plasma IGF-I concentrations were raised to the normal range for young adult men by the dose of growth hormone administered, with no evidence of tachyphylaxis or hormone resistance.

The dose, approximately 0. We conclude that in aging men with low plasma IGF-I concentrations hepatic responsiveness to human growth hormone is not impaired, and the decline in plasma IGF-I concentrations in such men results from growth hormone deficiency rather than growth hormone resistance.

The increase in plasma IGF-I levels that occurs when growth hormone is administered to children with growth hormone deficiency reflects not only augmented hepatic production of IGF-I, but also increased production of one of the binding proteins that transport IGF-I.

At the beginning of our study, adverse reactions to human growth hormone were thought to be unlikely because physiologic doses were being used.

Furthermore, similar or larger doses have not caused undesired reactions in children or young adults.

The magnitude of the increases in lean body mass and the decreases in adipose-tissue mass 8. Until now, the evidence for such a conclusion came only from short-term nitrogen-balance experiments.

Salomon et al. In rodents, the increase in lean body mass in response to growth hormone is due to increases in the volume of skeletal muscle, skin, liver, kidney, and spleen.

The reduction in adipose-tissue mass when children with growth hormone deficiency are treated with human growth hormone is associated with a redistribution of adipose tissue from abdominal to peripheral areas.

Biosynthetic human growth hormone had no detectable effect on the bone density of the radius or proximal femur in the aging men, but it increased the density of the lumbar vertebrae by about 1.

Although the decrease in bone density with advancing age in men may be due in part to diminished secretion of growth hormone, 1.

A similar interpretation applies to the lack of increase in the mandibular-height ratio. What organs are responsible for the increase in lean body mass, and do their functional capacities change as well?

Only when such questions are answered can the possible benefits of human growth hormone in the elderly be explored.

Since atrophy of muscle and skin contributes to the frailty of older people, the potential benefits of growth hormone merit continuing attention and investigation.

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Roux, J. Taillandier, F. Weryha, B. The Endocrinologist Medicine Studies 1. Stephen Campbell, Cassandra Szoeke. Journal of Pharmacy Practice and Research 39 Phytotherapy Research 23 Skin Ageprint.

Kurt J. Reyes, Homer J. Growth Hormone and Insulin Use and Abuse. Andreassen, I. Raymond, C. Kistorp, P. Hildebrandt, J.

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Dharmavarapu, Vivekananda Kedage, Kumar M. Upsala Journal of Medical Sciences Erika T. Schwartz, Kent Holtorf. Primary Care: Clinics in Office Practice Valter D.

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Micaela Schäfer Jung - Publications

Autobiographie und Handlungsautonomie in der ersten britischen Frauenbewegung. Zidorn C. La maggioranza dei dati proviene comunque dalla cartografia floristica in corso. Wirth H. In: Wilhalm T. Bernhard Klein Aldershot: Ashgate, Die klare Erkenntnis dieses unglaublichen Geschehens nimmt jungen Eltern den Atem.

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